Hair tissue mineral analysis (HTMA), is an analytical test which measures the mineral content of the hair. The sampled hair, obtained by cutting the first inch and one-half of growth closest to the scalp at the nape of the neck, is prepared in a licensed clinical laboratory through a series of chemical and high temperature digestive procedures. Testing is then performed using highly sophisticated detection equipment and methods to achieve the most accurate and precise results.

Hair is ideal tissue for sampling and testing. First, it can be cut easily and painlessly and can be sent to the lab without special handling requirements. Second, clinical results have shown that a properly obtained sample can give an indication of mineral status and toxic metal accumulation following long term or even acute exposure.

A HTMA reveals a unique metabolic world: intracellular activity, which cannot be seen through most other tests. This provides a blueprint of the biochemistry occurring during the period of hair growth and development.

Examples:

• Thirty to 40 days following an acute exposure, elevated serum levels of lead may be undetectable. This is due to the body removing the lead from the serum as a protective measure and depositing the metal into such tissues as the liver, bones, teeth and hair.
• Nutrient loss from the body can become so advanced that severe health conditions can develop without any appreciable changes noted in those same nutrient levels in a blood test.
• Symptoms of elemental deficiency can be present long before low levels can be detected in the serum.
• Excess sodium is associated with hypertension, but adequate amounts are required for normal health.

Hair is used as one of the tissues of choice by the Environmental Protection Agency in determining toxic metal exposure. A 1980 report from the E.P.A. stated that human hair can be effectively used for biological monitoring of the highest priority toxic metals. This report confirmed the findings of other studies in the U.S. and abroad, which concluded that human hair may be a more appropriate tissue than blood or urine for studying community exposure to some trace elements

There are many factors to take into consideration, such as:
Diet - Improper diet through high intake of refined and processed foods, alcohol and fad diets can all lead to a chemical imbalance. Even the nutrient content of a "healthy" diet can be inadequate, depending upon the soil in which the food was grown or the method in which it was prepared.
Stress - Physical or emotional stress can deplete the body of many nutrients while also reducing the capability to absorb and utilize many nutrients.
Medications - Both prescription and over-the-counter medications can deplete the body stores of nutrient minerals and/or increase the levels of toxic metals. These medications include diuretics, antacids, aspirin and oral contraceptives.
Pollution - From adolescence through adulthood the average person is continually exposed to a variety of toxic metal sources such as cigarette smoke (cadmium), hair dyes (lead), hydrogenated oils (nickel), anti-perspirants (aluminum), dental amalgams (mercury and cadmium), copper and aluminum cookware and lead-based cosmetics. These are just a few of the hundreds of sources which can contribute to nutrient imbalances and adverse metabolic effects.
Nutritional Supplements - Taking incorrect supplements or improper amounts of supplements can produce many vitamin and mineral excesses and/or deficiencies, contributing to an overall biochemical imbalance.
Inherited Patterns - A predisposition toward certain mineral imbalances, deficiencies and excesses can be inherited from parents.

Minerals interact not only with each other but also with vitamins, proteins, carbohydrates and fats. Minerals influence each of these factors, and they, in turn, influence mineral status. Minerals act as enzyme activators, and vitamins are synergistic to minerals as coenzymes. It is extremely rare that a mineral disturbance develops without a corresponding disturbance in the synergistic vitamin(s). It is also rare for a disturbance in the utilization or activity of a vitamin to occur without affecting a synergistic mineral(s). For example, vitamin C affects iron absorption and reduces copper retention. Boron and iron influence the status of vitamin B2. Vitamin B2 affects the relationship between calcium and magnesium. Vitamin B1 enhances sodium retention, B12 enhances iron and cobalt absorption, and vitamin A enhances the utilization of zinc, while antagonizing vitamins D and E. Protein intake will affect zinc status, etc. Therefore, evaluating mineral status provides good clues of vitamin status and requirements. Continuing research at Trace Elements involves the recognition of many synergistic and antagonistic interrelationships between minerals and vitamins.